1. You continue to perform CPR. You elect to call medical control and report the situation. Until you are sure that the outcome is going to be terminal you continue your efforts. What is the optimal ventilation rate you would provide now?
one ventilations every 30 seconds or after every 30 chest compressions.
one ventilations every 30 seconds or after every 30 chest compressions.
one ventilation every five seconds.
two ventilations every 15 seconds.
2. You check for a pulse and it is adequate with chest compressions. Why is the Sp02 not registering on your arrival?
Poor distal perfusion.
Cardiac arrest.
Cold extremities.
All of the above.
3. Carbon monoxide (CO) poisoning is common from:
diesel, propane and gasoline powered engines.
space heaters.
gasoline cooking equipment.
all of the above.
4. Levels of carbon monoxide can come from industrial and household pollution. Industrial plants can produce many types of toxic chemical levels and other harmful gases such as cyanide and formaldehyde, which are very toxic and lethal over time. Levels of CO above are considered harmful with several hours of exposure.
25 ppm
50 ppm
250 ppm
1,000 ppm
5. Symptoms of CO poisoning start with the common signs and symptoms such as:
Headaches.
Vertigo and hallucinations.
Flu-like symptoms.
All of the above.
6. CO levels of ppm can be lethal in two to three breaths or in less than several minutes of exposure.
1,000
2,500
12,800
100,000
7. This patient likely is suffering from severe hypoxia. CO poisoning causes the CO to combine with oxygen to form:
Carbon dioxide.
Sodium sulphide.
Carboxyhemoglobin.
Carbon trioxide.
8. The CO can also affect:
The myoglobin.
Respiratory and cardiac systems.
Mitochondrial cytochrome oxide.
All of the above.
9. During resuscitation the only antidote for CO poisoning would be to utilize:
Low flow oxygen over four to six hours.
High flow oxygen for 16 to 24 hours.
Nasal prongs for 24 to 48 hours.
Blow-by oxygen.
10. Higher levels of CO poisoning causes:
A fast heart rate.
Low blood pressure.
Cardiac arrhythmias.
All of the above.
11. As CO levels increase, is caused.
Central nervous system depression
Unconsciousness, respiratory arrest and cardiac arrest
Hallucination and pinpoint pupils
A and B only
All of the above
12. Chronic CO poisoning causes:
Myocardial ischemia, atrial fibrillation, pulmonary edema, pneumonia and high blood pressure.
Hyperglycemia, lactic acidosis, muscle necrosis and acute renal failure.
Auditory complications, dementia, psychosis and delayed neurological symptoms.
All of the above.
13. CO also binds to the hemeprotien myoglobin in muscle. It has a high affinity for myoglobin which is somewhere around to times greater than that of oxygen.
Two, Four
10, 25
25, 40
50, 60
14. The fire team is back and there are no other occupants in the house. You elect to transport immediately as there is a short transport time. Medical control advises you to continue resuscitation until first-line medications are administered or:
Until 10 minutes of good BLS care has been performed.
Until 20 minutes of good BLS and ALS care has been performed.
Until 40 minutes of good ALS care has been performed.
Until the patient is in a hospital setting.
15. The affinity between hemoglobin and CO is approximately 230 times stronger than the affinity between hemoglobin and oxygen so:
Oxygen binds to the hemoglobin better then carbon monoxide.
CO binds to hemoglobin in preference to oxygen if it has a choice.
Carbon dioxide binds to hemoglobin in preference to oxygen.
A and C only.
16. As you start to transport the patient you notice a fast visible wide complex rhythm on the monitor. You suspect it to be:
Ventricular fibrillation.
Ventricular tachycardia.
Pulseless electrical activity.
Idioventricular tachycardia.
17. Your first action would be to:
Check for a pulse.
Defibrillate at 200 joules biphasic.
Continue CPR at a rate of around 100 compressions per minute.
Administer epinephrine 1 mg IVP.
18. Following several minutes your partner successfully intubates the patient with a 7.5 cuffed endotracheal tube on his first attempt. During ventilations with the BVM you notice some foamy secretions coming up the endotracheal tube. You decide it is likely pulmonary edema, which can be seen in CO poisoning as well as many other complications. The patient still has a pulse now as well. Your best actions would be to:
Attempt to suction out the secretions as they appear as required.
Add a small amount of PEEP and continue to ventilate the patient as per normal.
Administer 40 mg of Lasix IVP immediately.
All of the above.
19. The patient’s BP comes back at around 60 systolic. The interventions that can help this would be to:
Administer fluid bolus of 20 ml/kg now.
Consider administration of vasopressor agents earlier.
Transport to a facility with hyperbaric capabilities.
All of the above.
20. The patient appears to have seizure-like movements now. What would be the recommended therapy to decrease these movements?
Ensure the airway is secure, ensure appropriate oxygenation and ensure end tidal C02 is at the accepted level of somewhere around 35 to 45 now.
Administer Versed 2.5 mg or Valium 2.5 mg or Ativan 2 mg IVP.
Contact medical control and decide if a loading dose of Dilantin might be warranted depending on your transport time. We know a loading dose of Dilantin is given over one hour, might have some antiarrhythmic properties and lessen the ventricular tachycardia.
All of the above.
21. The patient has runs of ventricular tachycardia now that is not perfusing when you checked. The therapies that can assist this would be:
Administer lidocaine 1 to 1.5 mg/kg and then start an infusion of 2 mg/minute
Administer amiodarone 150 mg over two to 10 minutes and then consider starting the loading dose over one hour.
Administer magnesium sulfate if you suspect the patient might be an alcoholic as it is indicated in this situation and 2 grams over two to three minutes.
All of the above are possible but pick one and go down that pathway as per your local medication protocols or as you are advised per medical direction.
22. Upon arrival at the hospital the patient has a portable chest X-ray done to verify tube location and for other medical complications. The CXR looks terrible and the emergency physician suspects the worst possibilities as a differential diagnosis. The differential diagnosis in this case might also demonstrate signs such as ___________________ on the patchy, poor looking X-ray.
Acute Respiratory Distress Syndrome
Bilateral pneumonia
Frank pulmonary edema
All of the above
23. Upon arrival the patient goes back into a pulseless wide complex tachycardia arrest. The arterial blood gas states the pH is 6.85 now. The next interventions should include:
Initiating CPR.
Administering sodium bicarbonate 1 mEq/kg IVP.
Defibrillating at 360 joules monophasic or 200 joules biphasic as per your local protocols.
All of the above.
24. After 30 minutes of recurrent episodes of pulseless ventricular tachycardia the patient goes into a PEA and then finally asystole. The differential diagnosis should include:
Hydrogen ion problems, hypoxia, hypothermia, hyperthermia, hypoglycemia, hyperkalemia and hypovolemia.
Tension pneumothorax, tablets such as TCAs, cardiac tamponade, toxicity of methanol, ethanol or other toxic substances.
Chronic hypoperfusion due to the carbon monoxide effects on the cardiac centre making it an ineffective pump, cerebral hypoxia that has irreversibly damaged the brain stem.
All are appropriate.
25. The physician in charge of the code, along with the EMS and ER staff, has done everything possible with everyone assisting on the code to the best of their capabilities. The physician uses the fast scan to check the heart now. There is no heart motion. The next appropriate action would be to:
Stop efforts and call the code now as there is no hope the patient will survive and walk out of ICU alive.
Continue the code until a family member can be located or decide the best actions of the code team.
Trial a run of transcutaneous pacing immediately.
All of the above.